The relationship between blood pressure and nerves is primarily controlled by the autonomic nervous system, particularly the sympathetic branch, which directly impacts blood pressure by regulating the constriction and dilation of blood vessels, meaning when nerves are stimulated, especially via the sympathetic system, blood pressure can rise due to vasoconstriction; conversely, when the sympathetic activity decreases, blood pressure can lower. 
 
Key points about the nerve-blood pressure connection:
Baroreceptors:
Specialized nerve cells located in the carotid arteries and aortic arch act as "pressure sensors" that constantly monitor blood pressure and send signals to the brain to adjust accordingly. 
 
Sympathetic nervous system activation:
When the body perceives stress or needs to respond quickly, the sympathetic nervous system triggers the release of norepinephrine, causing blood vessels to constrict and raising blood pressure. 
 
Parasympathetic nervous system:
The parasympathetic nervous system, the "rest and digest" branch, can counteract the effects of the sympathetic system by promoting vasodilation and lowering blood pressure. 
 
How this connection can be affected:
Stress:
Experiencing stress can trigger the sympathetic nervous system, causing a temporary spike in blood pressure. 
 
Anxiety disorders:
Chronic anxiety can lead to increased sympathetic activity, potentially contributing to high blood pressure. 
 
Medical conditions:
Certain medical conditions like autonomic dysfunction can disrupt the normal regulation of blood pressure through the nervous system.